Haulinippu on oikeassa veren vuotamisesta. Kuten teurastajat, metsämiehet, balsamoijat ja joulukinkun suolaajat hyvin tietävät, väkivaltaisesti kuolleiden veri ei (välttämättä) hyydy ollenkaan, varsinkaan jos ruumista liikutellaan. Oliko Annijatta, joka muisti sian verta ravistellun teurastuksen jälkeen että se pysyi juoksevana niin saatiin myöhemmin verilettuja.
Tässä linkkeja selityksiin ja vanhoihin vääntöihin asiasta:
http://murha.info/phpbb2/viewtopic.php? ... yy#p528408
http://murha.info/phpbb2/viewtopic.php? ... yy#p515167
Lividity is able to develop post mortem under the influence of gravity because the blood remains liquid rather than coagulating throughout the vascular system. Within about 30-60 minutes of death the blood in most corpses, dead from natural or non-natural causes, becomes permanently incoagulable. This is due to the release of fibrinolysins, especially from small calibre vessels, e.g. capillaries, and from serous surfaces, e.g. the pleura. Clots may persist when the mass of clot is too large to be liquified by the fibrinolysin available at the site of clot formation. In some deaths associated with infection and cachexia, this fibrinolytic effect may fail to develop, explaining the presence of abundant clot in the heart and large calibre vessels. Thus, in cases of sudden death the blood remains spontaneously coagulable only during a brief period immediately following death; it then becomes completely free from fibrinogen and will never again clot. This incoagulability of the blood is a commonplace observation at autopsy. The fluidity of the blood is not characteristic of any special cause or mechanism of death although many texts state that the blood remains liquid longer in asphyxial deaths
Abstract
Post-mortem blood may be either (a) semi-solidified, resembling clots or thrombi or their combinations, or entirely ‘white’ thrombi; or (b), in occasional cases, wholly liquid and incoagulable. Because such variations have confused observers, we will suggest an explanation for them in terms of the valve cusp hypoxia hypothesis (VCHH). The central question is whether it can rightly be imagined that blood coagulates after death. This was intensely debated in the early 20th century but was then resolved ad hoc. To attempt a resolution, the forensic and judicial implications of our account of the states of post-mortem blood will be explored and some clinical inferences drawn. This discussion will highlight the essential value of an a priori account of DVT aetiology.
http://link.springer.com/chapter/10.100 ... -6650-4_13
